By Celia Marr BVMS MVM PhD MRCVS, Mark Bowen BVetMed PhD CertEM(IntMed) MRCVS

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80. Keen JA, Hillier C, McGorum BC, Nally JE. Endothelin mediated contraction of equine laminar veins. Equine Vet J 2008;40(5): 488–492. 81. Eades SC, Stokes AM, Johnson PJ, LeBlanc CJ, Ganjam VK, Buff PR, et al. Serial alterations in digital hemodynamics and endothelin-1 immunoreactivity, platelet- 82. 83. 84. 85. 86. Chapter |2| neutrophil aggregation, and concentrations of nitric oxide, insulin, and glucose in blood obtained from horses following carbohydrate overload. Am J Vet Res 2007;68(1):87–94.

The reason why l-arginine boosts NO production by vessels undergoing atheromatous change is poorly understood and is the subject of intense investigation. Other therapeutic advances in the field of NO biology are aimed at inhibiting excessive NO production. The induction of iNOS can be inhibited by glucocorticoids, perhaps explaining the efficacy of these drugs if given in anticipation of septic shock. Selective iNOS inhibitors may be ideal in the treatment of septic shock, restoring vascular tone and arterial blood pressure.

J Cardiovasc Pharmacol 2006;47(1):46–50. 46. Konstam MA, Neaton JD, Poole-Wilson PA, et al. Comparison of losartan and captopril on heart failure-related outcomes and symptoms from the losartan heart failure survival study 32 47. 48. 49. 50. 51. 52. 53. 54. 55. (ELITE II). Am Heart J 2005; 150(1):123–131. Pitt B, Zannad F, Remme WJ, Cody R, Castaigne A, Perez A, et al. The effect of spironolactone on morbidity and mortality in patients with severe heart failure. Randomized Aldactone Evaluation Study Investigators.

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